The gene did their normal job of producing a protein that kills damaged cancer cells. But the researchers found that mice with an extra copy of the genes actually lived longer even when stripping out the impact of having less cancer.

　　This is the first anti-cancer gene tested for its effect on aging,Serrano said. The mice lived 16 percent longer in their average life span.

　　The p53 gene, when working properly, makes sure that damaged cells destroy themselves and do not divide uncontrollably to cause tumors. If the gene is mutated or inactivated the control mechanism does not work.

　　Serrano also said that other research has shown that mice and worms that eat less have slower metabolisms and live longer. But his study offers evidence that the mice can benefit from the extra copy of the genes without having to be starved.

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