If this does not kill the invader outright, then it acts as a flag for the bug in question to beeaten by a macrophage.
如果抗体不能马上杀死入侵者,则它将作为入侵者的一个标记,使得它们有可能被巨噬细胞所吞噬,
That happens when the antibody that is attached to the invader also attaches itself to anFc-receptor molecule.
这个过程只有当结合在入侵者上的抗体同时也能和Fc受体分子结合时才会发生,
This done, the invader is ingested.
而这个步骤一完成,则入侵者就被吞噬了。
Dr Chapmans plan was to use this mechanism to get macrophages to eat toxin molecules.
查普曼博士的计划是,利用这个机制,让巨噬细胞把肉毒素吞进去,
Once inside, they would disable SNAP-25 and thus stop the release of signalling moleculesin the way they do in nerve cells.
而进入细胞之后,它就能使SNAP-25失去活性,从而像在神经细胞中一样,阻止信号分子的释放。
The result, the theory goes, would be an end to the chronic inflammation that arthritiscauses, and thus to the chronic pain.
如果这个理论可行,那么由关节炎所导致的慢性炎症将被终结,同时被终结的,还有长期以来的痛苦。
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