那些不能释放肌肉抑制素的肥胖小白鼠依然保持肥胖,但同时也变得更加强壮。与瘦弱的对照小白鼠相比,它们的心脏和新陈代谢表现的更加健康。
They were in dramatically better shape than obese mice with unrestricted myostatin production.
与那些无限释放肌肉抑制素的小白鼠相比,这些小白鼠的体型要健康得多,这令人惊讶。
Dr Butcher added: "In our muscular obese mouse, despite full presentation of obesity, it appears that several of these key pathologies are prevented.
布彻博士补充道:“这些肌肉发达却肥胖的小白鼠,尽管依然表现出肥胖的症状,但一些关键的病理症状没有发生。”
"While much more research is needed, at this point myostatin appears to be a very promising pathway for protection against obesity-derived cardiometabolic dysfunction."
“虽然还需进行大量研究,但目前看来,肌肉抑制素似乎对于治疗由肥胖导致的心血管功能异常很有帮助。”
The research was presented at the American Physiological Society's annual meeting, Experimental Biology 2017, in Chicago, US.
该研究在美国芝加哥举办的美国生理学协会2017年实验生物学年会上作了报告。
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